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Botany 3% exam weight

Hemodynamic Disorders

Part of the INI CET (AIIMS PG) study roadmap. Botany topic pathol-004 of Botany.

Acute Inflammation is a high-yield inflammation topic for INI CET (AIIMS PG).

Definition: Inflammation is the protective vascular and tissue response to injury, aimed at removing the injurious agent and limiting tissue damage.

Cardinal Signs of Inflammation (Celsus’s signs):

  1. Rubor — Redness (vasodilation)
  2. Tumor — Swelling (edema and cellular infiltration)
  3. Calor — Heat (increased blood flow)
  4. Dolor — Pain (sensation from chemical mediators)
  5. Functio laesa — Loss of function (added by Galen)

Two Types of Inflammation:

Acute Inflammation: Rapid onset (seconds to minutes), short duration (hours to days)

  • Features: Vasodilation, increased vascular permeability, neutrophil emigration, exudate formation
  • Exudate: High protein, high cellular content — indicates significant tissue injury
  • Transudate: Low protein, low cellular content — caused by hydrostatic/oncotic pressure changes

Chronic Inflammation: Longer duration (weeks to months), characterized by:

  • Lymphocyte and macrophage infiltration
  • Fibrosis and angiogenesis
  • Granuloma formation

Chemical Mediators of Inflammation:

MediatorSourceEffect
HistamineMast cells, basophils, plateletsVasodilation, increased permeability
ProstaglandinsArachidonic acid (COX pathway)Pain, fever, vasodilation
LeukotrienesArachidonic acid (LOX pathway)Bronchoconstriction, vascular permeability
Cytokines (TNF-α, IL-1)MacrophagesFever, systemic acute-phase response
Complement (C3a, C5a)Liver (hepatocytes)Anaphylatoxins, chemotaxis
Kinins (bradykinin)PlasmaPain, vasodilation, permeability

Exam Tip for INI CET (AIIMS PG): NSAIDs block COX pathway → reduce prostaglandins → reduce pain, fever, and inflammation. This is why they’re called anti-inflammatory.

Outcomes of Inflammation:

  1. Complete resolution (ideal outcome)
  2. Healing by fibrosis (scarring)
  3. Chronic inflammation
  4. Suppuration (abscess formation)
  5. Progression to necrosis